Pre_GI: SWBIT SVG BLASTP

Query: NC_014006:850763 Sphingobium japonicum UT26S chromosome 1, complete genome

Lineage: Sphingobium japonicum; Sphingobium; Sphingomonadaceae; Sphingomonadales; Proteobacteria; Bacteria

General Information: Sphingobium japonicum (strain NBRC 101211 / UT26S) is a yellow-pigmented, non-motile, aerobic, hexachlorocyclohexane (HCH)-degrading Gram-negative bacterium isolated from a dumpsite in the vicinity of an industry that has been producing lindane. HCH has been used extensively as insect control agents in the health and agriculture fields. Sphingobium japonicum, formerly Sphingomonas paucimobilis, is able to degrade hexachlorocyclohexane which has been used extensively as insect control agents in the health and agriculture fields.

- Sequence; - BLASTP hit: hover for score (Low score = Light, High score = Dark);
- hypothetical protein; - cds: hover for description

BLASTP Alignment.txt

Subject: NC_010102:2322866 Salmonella enterica subsp. enterica serovar Paratyphi B str. SPB7,

Lineage: Salmonella enterica; Salmonella; Enterobacteriaceae; Enterobacteriales; Proteobacteria; Bacteria

General Information: This strain (SGSC 4150; ATCC BAA-1250) was isolated from a stool sample of an infected woman in Penang, Malaysia, May 16, 2002. This strain is susceptible to antibiotics, and was classified as serovar Paratyphi B because it was unable to metabolize D-tartrate. Causes enteric infections. This group of Enterobactericiae have pathogenic characteristics and are one of the most common causes of enteric infections (food poisoning) worldwide. They were named after the scientist Dr. Daniel Salmon who isolated the first organism, Salmonella choleraesuis, from the intestine of a pig. The presence of several pathogenicity islands (PAIs) that encode various virulence factors allows Salmonella spp. to colonize and infect host organisms. There are two important PAIs, Salmonella pathogenicity island 1 and 2 (SPI-1 and SPI-2) that encode two different type III secretion systems for the delivery of effector molecules into the host cell that result in internalization of the bacteria which then leads to systemic spread.