Query: NC_011026:1284966 Chloroherpeton thalassium ATCC 35110, complete genome Lineage: Chloroherpeton thalassium; Chloroherpeton; Chlorobiaceae; Chlorobiales; Chlorobi; Bacteria General Information: Chloroherpeton thalassium is a non-filamentous, flexing and gliding green sulfur bacterium isolated from marine sources off the North East coast of the USA in the 1980s. This organism is the most distant member of the green sulfur bacteria and is commonly found in aquatic microbial mats. Chloroherpeton thalassium is distinct from the other green sulfur bacteria in that it grows as a filamentous rod and is capable of gliding motility. Chloroherpeton thalassium is a photolithotroph, obtaining energy by anoxygenic photosynthesis using sulfide or sulfur as the electron donor. The photosynthetic apparatus consist of membrane-bound iron/sulfur reaction center associated with a light absorbing organelle, the chlorosome.
- Sequence; - BLASTP hit: hover for score (Low score = Light, High score = Dark); - hypothetical protein; - cds: hover for description
General Information: Endosymbiont. Obligate intracellular bacterium infects around 20% of all insect species. Naturally infects Drosophila simulans and induces almost complete cytoplasmic incompatibility in its host. Wolbachia sp. subsp. Drosophila simulans (strain wRi) is an intracellular proteobacterium that infect insects as well as isopods, spiders, scorpions, mites, and filarial nematodes. It is maternally inherited and induces reproductive alterations of insect populations by male killing, feminization, parthenogenesis, or cytoplasmic incompatibility. In insect populations, Wolbachia sp. induce reproductive manipulations to enhance their own spreading. The most frequently observed reproductive abnormality is cytoplasmic incompatibility, where uninfected females are unable to produce offspring with infected males, whereas infected females can produce offspring with both infected and uninfected males, thus creating a reproductive advantage for infected females. Other spectacular effects of Wolbachia sp. infections are male embryo killing, feminization, and parthenogenesis induction.