Pre_GI: SWBIT SVG BLASTP

Query: NC_011004:5283648 Rhodopseudomonas palustris TIE-1, complete genome

Lineage: Rhodopseudomonas palustris; Rhodopseudomonas; Bradyrhizobiaceae; Rhizobiales; Proteobacteria; Bacteria

General Information: This strain was isolated from an iron-rich microbial mat from School Street Marsh in Woods Hole, MA, USA. It grows photoautotrophically with Fe(II), H2, or thiosulfate as the electron donor, photoheterotrophically with a variety of organic carbon sources and chemoheterotrophically in the dark. This organism has a diverse metabolism and is capable of growth using light, inorganic, or organic compounds as energy sources and carbon dioxide or organic compounds as carbon sources. Commonly found in soil and water environments this bacterium is also capable of degrading a wide range of toxic organic compounds, and may be of use in bioremediation of polluted sites. The bacterium undergoes differentiation to produce a stalked nonmotile cell and a motile flagellated cell. In the presence of light, this bacterium produces a number of intracellular membranous vesicles to house the photosynthetic reaction centers.

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BLASTP Alignment.txt

Subject: NC_008570:2014151 Aeromonas hydrophila subsp. hydrophila ATCC 7966, complete genome

Lineage: Aeromonas hydrophila; Aeromonas; Aeromonadaceae; Aeromonadales; Proteobacteria; Bacteria

General Information: Isolated from canned milk from the USA. Aquatic organism that can cause foodborne illnesses in humans. This organism is an enviromental bacterium that is often found in aquatic habitats, but can also be found contaminating food products. It causes a variety of diseases in both cold-blooded and warm-blooded organisms. The bacterium is becoming a problematic pathogen in humans, where it causes gastroenteritis and septicemia, mainly due to the development of antibiotic resistance by this organism. One of the major virulence factors is aerolysin, a toxin that is produced and secreted by the cell via a type II secretion apparatus. Other virulence functions include a surface layer which inhibits complement-mediated killing, type IV pili for attachment, and a set of extracellular proteases which can cause tissue damage.