Pre_GI: SWBIT SVG BLASTP

Query: NC_008513:343540 Buchnera aphidicola str. Cc (Cinara cedri), complete genome

Lineage: Buchnera aphidicola; Buchnera; Enterobacteriaceae; Enterobacteriales; Proteobacteria; Bacteria

General Information: This strain is found in the cedar aphid, Cinara cedri. Aphid endosymbiont. Almost all aphids contain maternally transmitted bacteriocyte cells, which themselves contain bacteria called Buchnera. The aphids live on a restricted diet (plant sap), rich in carbohydrates, but poor in nitrogenous or other essential compounds. It is believed that the Buchnera provide the essential nutrients the host lacks. Besides a nutritional co-dependence, due to a co-existence of millions of years, Buchnera have lost the ability to produce cell surface components such as lipopolysaccharides. This makes for an obligate endosymbiont relationship between host and Buchnera. Buchnera are prokaryotic cells which belong to the gamma-Proteobacteria, closely related to the Enterobacteriaceae family. Phylogenetic studies using 16S rRNA indicate that the symbiotic relationship was established around 200-250 million years ago. Since Buchnera are closely related to Escherichia coli and Haemophilus influenzae, comparative genomic studies can shed light on the evolutionary mechanisms of intracellular endosymbiosis as well as the different underlying molecular basis between organisms with parasitic behavior and symbionts.

Subject: NC_003919:5056650 Xanthomonas axonopodis pv. citri str. 306, complete genome

Lineage: Xanthomonas citri; Xanthomonas; Xanthomonadaceae; Xanthomonadales; Proteobacteria; Bacteria

General Information: This organism is the causal agent of citrus canker, a bacterial infection originating from southeast Asia which now occurs worldwide. Primarily a pathogen of plants in the Citrus genus, the disease is sometimes also found in other members of the Rutaceae. The bacterium survives in leaf, shoot and fruit lesions that develop during the spring, and which also cause secondary infections. During warm, wet weather in spring and early summer, the bacterium oozes out of overwintering lesions and infects new growth via the stomal pores or wounds. The bacterium may also survive for various periods of time in the soil or associated with other hosts.