Pre_GI: SWBIT SVG BLASTP

Query: NC_007643:889755 Rhodospirillum rubrum ATCC 11170, complete genome

Lineage: Rhodospirillum rubrum; Rhodospirillum; Rhodospirillaceae; Rhodospirillales; Proteobacteria; Bacteria

General Information: This bacterium can grow using carbon monoxide as the sole carbon and energy source and the cells contain a well characterized nitrogenase system that is post-translationally modified by ADP-ribosylation. Phototrophic bacterium. This organism is an anoxygenic phototrophic bacterium that does not produce oxygen, but instead produces extracellular elemental sulfur when harvesting light energy. The bacterium lacks the light harvesting complex 2 (LHC2) normally found in photosynthetic bacteria meaning it contains one of the simplest photosynthetic systems studied.

- Sequence; - BLASTP hit: hover for score (Low score = Light, High score = Dark);
- hypothetical protein; - cds: hover for description

BLASTP Alignment.txt

Subject: NC_008599:596849 Campylobacter fetus subsp. fetus 82-40, complete genome

Lineage: Campylobacter fetus; Campylobacter; Campylobacteraceae; Campylobacterales; Proteobacteria; Bacteria

General Information: This strain (82-40) was isolated from the blood of a human patient who was having a renal transplant and is the best characterized isolate of this species.. The ratio of bloodstream infection to diarrheal illnesses for C. fetus is nearly 400-fold higher than for C. jejuni, indicating its marked propensity for invasive disease compared to C. jejuni. Causes infertility, infectious abortions in cattle, opportunistic human pathogen. This organism causes infertlity and infectious abortions in domesticated sheep, goats and cattle. It is an opportunistic pathogen in humans which can severely affect immunocompromised patients. Initially the bacterium can cause gastroenteritis, and then spread systemically throughout the blood (bacteremia) and cause septicemia, meningitis, and other systemic infections. This layer is essential for host colonization, and prevents complemented-mediated immune responses by inhibiting complement C3b binding.