Pre_GI: SWBIT SVG BLASTN

Query: NC_014259:3369000 Acinetobacter sp. DR1 chromosome, complete genome

Lineage: Acinetobacter oleivorans; Acinetobacter; Moraxellaceae; Pseudomonadales; Proteobacteria; Bacteria

General Information: This strain is capable of degrading paraffin, n-hexadecane, diesel. Acinetobacter sp. DR1 is a diesel degrader Gram-negative bacterium isolated from the soil of rice paddies in Soth Korea. They are found in many different habitats, including soil, water, and sewage, as well as in a variety of foodstuffs. They are also members of the normal microflora of human skin, and have been implicated as a presumed causative or contributory agent of numerous nosocomial (in-hospital) infections, especially those in immunocompromised individuals. Due to their genomic versatility, these bacteria are highly adaptive to treatment with various antibiotic reagents.

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Subject: NC_012416:979484 Wolbachia sp. wRi, complete genome

Lineage: Wolbachia; Wolbachia; Anaplasmataceae; Rickettsiales; Proteobacteria; Bacteria

General Information: Endosymbiont. Obligate intracellular bacterium infects around 20% of all insect species. Naturally infects Drosophila simulans and induces almost complete cytoplasmic incompatibility in its host. Wolbachia sp. subsp. Drosophila simulans (strain wRi) is an intracellular proteobacterium that infect insects as well as isopods, spiders, scorpions, mites, and filarial nematodes. It is maternally inherited and induces reproductive alterations of insect populations by male killing, feminization, parthenogenesis, or cytoplasmic incompatibility. In insect populations, Wolbachia sp. induce reproductive manipulations to enhance their own spreading. The most frequently observed reproductive abnormality is cytoplasmic incompatibility, where uninfected females are unable to produce offspring with infected males, whereas infected females can produce offspring with both infected and uninfected males, thus creating a reproductive advantage for infected females. Other spectacular effects of Wolbachia sp. infections are male embryo killing, feminization, and parthenogenesis induction.