Query: NC_013892:1503162 Xenorhabdus bovienii SS-2004 chromosome, complete genome Lineage: Xenorhabdus bovienii; Xenorhabdus; Enterobacteriaceae; Enterobacteriales; Proteobacteria; Bacteria General Information: Insect pathogenic, facultatively anaerobic, non-spore forming Gram-negative bacterium. This genus is a group of insect pathogens which live in a mutualistic relationship with the soil nematode family Steinernematidae. Free-living, juvenile Steinernema spp. enter insect larvae through the digestive tract. They penetrate the larvae body cavity and release Xenorhabdus spp. into the hemolymph (blood). The bacteria multiply rapidly, killing the larvae, and providing suitable nutrient conditions for the growth and reproduction of the Steinernema spp. The nematode matures and reproduces. The new juveniles reassociate with Xenorhabdus spp. and are released into the soil.Xenorhabdus bovienii. Unlike Xenorhabdus nematophila, which is found in only in Steinernema carpocapsae, Xenorhabdus bovienii is associated with several different species of Steinernema.
- Sequence; - BLASTN hit (Low score = Light, High score = Dark) - hypothetical protein; - cds: hover for description
General Information: Salmonella enterica subsp. enterica serovar Agona causes gastroenteritis in humans and is also pathogenic to swine and other food animals. This serovar is able to contain the Salmonella genomic island 1 multidrug resistance gene cluster. This group of Enterobactericiae have pathogenic characteristics and are one of the most common causes of enteric infections (food poisoning) worldwide. They were named after the scientist Dr. Daniel Salmon who isolated the first organism, Salmonella choleraesuis, from the intestine of a pig. The presence of several pathogenicity islands (PAIs) that encode various virulence factors allows Salmonella spp. to colonize and infect host organisms. There are two important PAIs, Salmonella pathogenicity island 1 and 2 (SPI-1 and SPI-2) that encode two different type III secretion systems for the delivery of effector molecules into the host cell that result in internalization of the bacteria which then leads to systemic spread.