Pre_GI: SWBIT SVG BLASTN

Query: NC_011184:325797 Vibrio fischeri MJ11 chromosome I, complete sequence

Lineage: Aliivibrio fischeri; Aliivibrio; Vibrionaceae; Vibrionales; Proteobacteria; Bacteria

General Information: This strain was isolated from a pinecone fish, Monocentris japonica, light-emitting organs in Japan. This genus is abundant in marine or freshwater environments such as estuaries, brackish ponds, or coastal areas; regions that provide an important reservoir for the organism in between outbreaks of the disease. Vibrio can affect shellfish, finfish, and other marine animals and a number of species are pathogenic for humans. This organism is found in marine environments and was originally named by Bernard Fischer during a sea voyage in the 1800s. It is a symbiont in fish and squids and is responsible for light generation in those organisms, which use it as a defense mechanism to avoid predators.

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BLASTN Alignment.txt

Subject: NC_008599:596849 Campylobacter fetus subsp. fetus 82-40, complete genome

Lineage: Campylobacter fetus; Campylobacter; Campylobacteraceae; Campylobacterales; Proteobacteria; Bacteria

General Information: This strain (82-40) was isolated from the blood of a human patient who was having a renal transplant and is the best characterized isolate of this species.. The ratio of bloodstream infection to diarrheal illnesses for C. fetus is nearly 400-fold higher than for C. jejuni, indicating its marked propensity for invasive disease compared to C. jejuni. Causes infertility, infectious abortions in cattle, opportunistic human pathogen. This organism causes infertlity and infectious abortions in domesticated sheep, goats and cattle. It is an opportunistic pathogen in humans which can severely affect immunocompromised patients. Initially the bacterium can cause gastroenteritis, and then spread systemically throughout the blood (bacteremia) and cause septicemia, meningitis, and other systemic infections. This layer is essential for host colonization, and prevents complemented-mediated immune responses by inhibiting complement C3b binding.