Query: NC_009925:4997000 Acaryochloris marina MBIC11017, complete genome Lineage: Acaryochloris marina; Acaryochloris; ; Chroococcales; Cyanobacteria; Bacteria General Information: Acaryochloris marina MBIC11017 was isolated from algae from the coast of the Palau Islands in the western Pacific. Marine cyanobacterium. Acaryochloris marina was first isolated as an epiphyte of algae. M. marina been isolated from a variety of habitats and locations, usually associated with algae but also as free-living organisms. This cyanobacterium produces an atypical photosynthetic pigment, chlorophyll d, as the major reactive agent. The oxygenic photosynthesis based on this pigment may have evolved as an acclimatization to far-red light environments, or an as intermediate between the red-absorbing oxygenic and the far-red-absorbing anoxygenic photosynthesis that uses bacteriochlorophylls. Because of the unusual ratio of chlorophyll a to chlorophyll d in this organism, it has been used as a model to study the spectrographic characteristics of the two pigments.
- Sequence; - BLASTN hit (Low score = Light, High score = Dark) - hypothetical protein; - cds: hover for description
General Information: This strain (82-40) was isolated from the blood of a human patient who was having a renal transplant and is the best characterized isolate of this species.. The ratio of bloodstream infection to diarrheal illnesses for C. fetus is nearly 400-fold higher than for C. jejuni, indicating its marked propensity for invasive disease compared to C. jejuni. Causes infertility, infectious abortions in cattle, opportunistic human pathogen. This organism causes infertlity and infectious abortions in domesticated sheep, goats and cattle. It is an opportunistic pathogen in humans which can severely affect immunocompromised patients. Initially the bacterium can cause gastroenteritis, and then spread systemically throughout the blood (bacteremia) and cause septicemia, meningitis, and other systemic infections. This layer is essential for host colonization, and prevents complemented-mediated immune responses by inhibiting complement C3b binding.