Query: NC_009138:2110500 Herminiimonas arsenicoxydans, complete genome Lineage: Herminiimonas arsenicoxydans; Herminiimonas; Oxalobacteraceae; Burkholderiales; Proteobacteria; Bacteria General Information: Herminiimonas arsenicoxydans was isolated from heavy metal contaminated sludge from an industrial water treatment plant. This organism has a number of mechanisms for metabolizing arsenic allowing it to effectively colonize arsenic-contaminated environments. A bacterium capable of oxidizing and reducing arsenic. This heterotrophic bacterium is capable of reducing and oxidizing arsenic with the objective of detoxification. Arsenic is both a product from natural sources and of human activities, and is widely distributed in the environment, essentially in 3 different oxidation states: As (-III) (arsine), As (+III) (arsenite) and As (+V) (arseniate). The ecology of this metalloid is strongly dependent on microbial transformations which affect the mobility and bioavailability as well as the toxicity of arsenic in the environment.
- Sequence; - BLASTN hit (Low score = Light, High score = Dark) - hypothetical protein; - cds: hover for description
General Information: This organism was discovered in 1819 by Bizio who named the organism after the Italian physicist Serrati. It was considered a nonpathogenic organism until late in the 20th century, although pathogenicity was noted as early as 1913. Serratia marcescens is an opportunistic human pathogen that is increasingly associated with life-threatening hospital-acquired infections. It is an environmental organism that has a broad host range, and is capable of infecting vertebrates and invertebrates, as well as plants. In humans, Serratia marcescens can cause meningitis (inflammation of the membrane surrounding the brain and spinal cord), endocarditis (inflammation of heart muscle) and pyelonephritis (inflammation of the kidneys). Many strains are resistant to multiple antibiotics. Environmental isolates are noted by production of the red pigment prodigiosin.