Pre_GI: SWBIT SVG BLASTN

Query: NC_008752:2056547 Acidovorax avenae subsp. citrulli AAC00-1, complete genome

Lineage: Acidovorax citrulli; Acidovorax; Comamonadaceae; Burkholderiales; Proteobacteria; Bacteria

General Information: This organism is the causal agent of bacterial fruit blotch, which was first detected in Florida in 1989. The disease is spread by infested seed, infected transplants, or natural spread from wild hosts. Infected transplants represent the most important means of disease transmission because fruit blotch can spread throughout the transplant operation and can be asymptomatic on older plants, which can lead to high numbers of infected young plants early in the planting season.

- Sequence; - BLASTN hit (Low score = Light, High score = Dark)
- hypothetical protein; - cds: hover for description

BLASTN Alignment.txt

Subject: NC_020207:2240115 Enterococcus faecium NRRL B-2354, complete genome

Lineage: Enterococcus faecium; Enterococcus; Enterococcaceae; Lactobacillales; Firmicutes; Bacteria

General Information: This genera consists of organisms typically found in the intestines of mammals, although through fecal contamination they can appear in sewage, soil, and water. They cause a number of infections that are becoming increasingly a problem due to the number of antibiotic resistance mechanisms these organisms have picked up. Both Enterococcus faecalis and Enterococcus faecium cause similar diseases in humans, and are mainly distinguished by their metabolic capabilities. This opportunistic pathogen causes a range of infections similar to those observed with Enterococcus faecalis, including urinary tract infections, bacteremia (bacteria in the blood), and infective endocarditis (inflammation of the membrane surrounding the heart). Hospital-acquired infections from this organism are on the rise due to the emergence of antiobiotic resistance strains and has led to the rise of vancomycin-resistant Staphylococcus aureus strains due to the horizontal transfer of Enterococcus antibiotic resistance genes. Little is known about the virulence mechanisms in this organism, but the genome does encode an esp gene for the surface adhesin. Vancomycin resistant isolates are more typically Enterococcus faecium than Enterococcus faecalis.