Pre_GI: SWBIT SVG BLASTN

Query: NC_006677:1596560 Gluconobacter oxydans 621H, complete genome

Lineage: Gluconobacter oxydans; Gluconobacter; Acetobacteraceae; Rhodospirillales; Proteobacteria; Bacteria

General Information: Industrially useful bacterium. Gluconobacter oxydans is a member of the Acetobacteraceae family within the alpha proteobacteria and can be isolated from flowers, fruits, and fermented beverages. This organism uses membrane-associated dehydrogenases to incompletely oxidize a wide variety of carbohydrates and alcohols. Oxidation occurs in the periplasm with the products being released into the medium via outer membrane porins and the electrons entering the electron transport chain. Able to oxidize large amounts of substrates, making it useful for industrial purposes. Among other applications, it has been used to produce 2-ketogluconic for iso-ascorbic acid production, 5-ketogluconic acid from glucose for tartaric acid production, and L-sorbose from sorbitol for vitamin C synthesis.

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BLASTN Alignment.txt

Subject: NC_008599:596849 Campylobacter fetus subsp. fetus 82-40, complete genome

Lineage: Campylobacter fetus; Campylobacter; Campylobacteraceae; Campylobacterales; Proteobacteria; Bacteria

General Information: This strain (82-40) was isolated from the blood of a human patient who was having a renal transplant and is the best characterized isolate of this species.. The ratio of bloodstream infection to diarrheal illnesses for C. fetus is nearly 400-fold higher than for C. jejuni, indicating its marked propensity for invasive disease compared to C. jejuni. Causes infertility, infectious abortions in cattle, opportunistic human pathogen. This organism causes infertlity and infectious abortions in domesticated sheep, goats and cattle. It is an opportunistic pathogen in humans which can severely affect immunocompromised patients. Initially the bacterium can cause gastroenteritis, and then spread systemically throughout the blood (bacteremia) and cause septicemia, meningitis, and other systemic infections. This layer is essential for host colonization, and prevents complemented-mediated immune responses by inhibiting complement C3b binding.