Pre_GI: SWBIT SVG BLASTN

Query: NC_004547:1062410 Erwinia carotovora subsp. atroseptica SCRI1043, complete genome

Lineage: Pectobacterium atrosepticum; Pectobacterium; Enterobacteriaceae; Enterobacteriales; Proteobacteria; Bacteria

General Information: This strain (ATCC BAA-672) is a virulent blackleg isolated from the stem of a potato plant. Causative agent for blackleg and soft rot disease in potatoes. Formerly Erwinia, these organisms are plant-specific pathogens that invade the vascular systems of plants. Both Pectobacterium chrysanthemia and Pectobacterium carotovora cause soft-rot diseases of various plant hosts through degradation of the plant cell walls. Pectobacterium colonize the intercellular spaces of plant cells and deliver potent effector molecules (Avr - avirulence) through a type III secretion system (Hrp - hypersensitive response and pathogenicity). Avr proteins control host-bacterium interactions, including host range. Expression of the plant cell-wall-degrading enzymes is controlled through a quorum-sensing mechanism that quantifies the number of Pectobacterium bacteria through measurement of the concentration of small molecules (acyl homoserine lactones) produced by Pectobacterium. Pectobacterium atrosepticum is an environmentally widespread organism that causes blackleg and soft rot disease in potatoes. This organism produces pectolytic enzymes that destroy plant tissue and allow the bacteria to spread.

Subject: NC_003919:103000 Xanthomonas axonopodis pv. citri str. 306, complete genome

Lineage: Xanthomonas citri; Xanthomonas; Xanthomonadaceae; Xanthomonadales; Proteobacteria; Bacteria

General Information: This organism is the causal agent of citrus canker, a bacterial infection originating from southeast Asia which now occurs worldwide. Primarily a pathogen of plants in the Citrus genus, the disease is sometimes also found in other members of the Rutaceae. The bacterium survives in leaf, shoot and fruit lesions that develop during the spring, and which also cause secondary infections. During warm, wet weather in spring and early summer, the bacterium oozes out of overwintering lesions and infects new growth via the stomal pores or wounds. The bacterium may also survive for various periods of time in the soil or associated with other hosts.